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Valdivielso, JoséM., Reverte, María, Rivas-Cabañero, Lina, López-Novoa, JoséM. (1996) Increased severity of gentamicin nephrotoxicity in aging rats is mediated by a reduced glomerular nitric oxide production. Environmental Toxicology and Pharmacology, 2. 73-75 doi:10.1016/1382-6689(96)00034-8

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Reference TypeJournal (article/letter/editorial)
TitleIncreased severity of gentamicin nephrotoxicity in aging rats is mediated by a reduced glomerular nitric oxide production
JournalEnvironmental Toxicology and Pharmacology
AuthorsValdivielso, JoséM.Author
Reverte, MaríaAuthor
Rivas-Cabañero, LinaAuthor
López-Novoa, JoséM.Author
Year1996 (August)Volume2
PublisherElsevier BV
DOIdoi:10.1016/1382-6689(96)00034-8Search in ResearchGate
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Mindat Ref. ID9727340Long-form Identifiermindat:1:5:9727340:6
GUID0
Full ReferenceValdivielso, JoséM., Reverte, María, Rivas-Cabañero, Lina, López-Novoa, JoséM. (1996) Increased severity of gentamicin nephrotoxicity in aging rats is mediated by a reduced glomerular nitric oxide production. Environmental Toxicology and Pharmacology, 2. 73-75 doi:10.1016/1382-6689(96)00034-8
Plain TextValdivielso, JoséM., Reverte, María, Rivas-Cabañero, Lina, López-Novoa, JoséM. (1996) Increased severity of gentamicin nephrotoxicity in aging rats is mediated by a reduced glomerular nitric oxide production. Environmental Toxicology and Pharmacology, 2. 73-75 doi:10.1016/1382-6689(96)00034-8
In(n.d.) Environmental Toxicology and Pharmacology Vol. 2. Elsevier BV

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Valdivielso, J.M, Rivas-Cabañero, L, Pérez-Barriocanal, F, López-Novoa, J.M (1997) Effect of nitric oxide synthesis modification on renal function in gentamicin-induced nephrotoxicity. Environmental Toxicology and Pharmacology, 3. 123-128 doi:10.1016/s1382-6689(97)00148-8
Wong, Donna, Qiu, Bo S., Ko, Joshua K.S., Koo, Marcel W.L., Cho, Chi H. (1996) Mucosal nitric oxide is not responsible for the hemodynamic changes induced by nicotine in rat stomachs. Environmental Toxicology and Pharmacology, 1. 167-174 doi:10.1016/1382-6689(96)00003-8
Marzzullo, Luciane, Oliveira, Edilberto P., Souza, Liliete C., Campa, Ana (1996) Toxicity of amphotericin B to analbuminemic rats. Environmental Toxicology and Pharmacology, 2. 31-34 doi:10.1016/1382-6689(96)00026-9
Gupta, Alka, Agarwal, Ashok K, Shukla, Girja S (2000) Effect of quinalphos and cypermethrin exposure on developing blood–brain barrier: role of nitric oxide. Environmental Toxicology and Pharmacology, 8. 73-78 doi:10.1016/s1382-6689(99)00034-4
Stella, Luigi, de Novellis, Vito, Berrino, Liberato, D'Amico, Michele, Rossi, Francesco (1996) Evidence that A2a and not A2b purinoceptors are coupled to production of nitric oxide in the central regulation of blood pressure. Environmental Toxicology and Pharmacology, 2. 327-329 doi:10.1016/s1382-6689(96)00065-8
(1996) Editorial Board. Environmental Toxicology and Pharmacology, 2. doi:10.1016/s1382-6689(96)90008-3
Kuo, Tsun-Cheng, Lin-Shiau, Shoei-Yn (1996) Activation of functionally protective K+ channels by methylmercury in rat alveolar macrophages. Environmental Toxicology and Pharmacology, 2. 9-15 doi:10.1016/1382-6689(96)00022-1
Tang, Hai-Wang, Hu, Xiao-Hong, Liang, You-Xin (1996) Alterations of striatal monoamine metabolites in young rats following pre- and postnatal lead exposure. Environmental Toxicology and Pharmacology, 1. 147-153 doi:10.1016/1382-6689(96)00002-6
Dayan, A.D. (1996) The immune response is subject to individual variability at all levels. Environmental Toxicology and Pharmacology, 2. 177-180 doi:10.1016/s1382-6689(96)00051-8
Eriksson, Per, Fredriksson, Anders (1996) Neonatal exposure to 2,2′,5,5′-tetrachlorobiphenyl causes increased susceptibility in the cholinergic transmitter system at adult age. Environmental Toxicology and Pharmacology, 1. 217-220 doi:10.1016/1382-6689(95)00017-8
Tohyama, Chiharu, Satoh, Masahiko, Kodama, Naomi, Nishimura, Hisao, Choo, Andy, Michalska, Anna, Kanayama, Yoshitaka, Naganuma, Akira (1996) Reduced retention of cadmium in the liver of metallothionein-null mice. Environmental Toxicology and Pharmacology, 1. 213-216 doi:10.1016/1382-6689(96)00014-2
 
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